Conolidine’s analgesic outcomes stem from its interaction with non-opioid pain pathways. Not like opioids, which bind to µ-opioid receptors during the central nervous system, conolidine modulates alternate molecular targets. A Science Advances analyze discovered that conolidine interacts Using the atypical chemokine receptor ACKR3/CXCR7, which regulates opioid peptide availability. By https://proleviate-conolidine68751.blogoxo.com/37825333/considerations-to-know-about-alternative-natural-pain-relief-to-replace-traditional-painkillers